A panel of 6 human glioma cell lines was examined for TGF-1 responsiveness. U-178 MG and U-251 MG AgCl1 were significantly inhibited by TGF-1, while U-343 MGa 31L and U-343 MGa 35L were potently stimulated to proliferate. TGF-1 induced endogenous PAI-1 protein synthesis, Smad binding element/(CAG
Expression of TGF-β isoforms, TGF-β receptors, and SMAD molecules at different stages of human glioma
✍ Scribed by Christian Kjellman; Sabine P. Olofsson; Oscar Hansson; Torbjörn Von Schantz; Magnus Lindvall; Ingar Nilsson; Leif G. Salford; Hans-Olov Sjögren; Bengt Widegren
- Publisher
- John Wiley and Sons
- Year
- 2000
- Tongue
- French
- Weight
- 170 KB
- Volume
- 89
- Category
- Article
- ISSN
- 0020-7136
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