𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Expression of receptors for plasminogen activators on endothelial cell surface depends on their origin

✍ Scribed by H. C. Kwaan; J. Wang; I. Weiss


Book ID
109154987
Publisher
John Wiley and Sons
Year
2004
Tongue
English
Weight
125 KB
Volume
2
Category
Article
ISSN
1538-7933

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Dependence of human vascular cell surfac
✍ Hirofumi Sawa; Craig H. Lundgren; Steven L. Brown; Satoshi Fujii πŸ“‚ Article πŸ“… 1996 πŸ› Springer US 🌐 English βš– 784 KB

To delineate the role of binding of urokinase type plasminogen activator (uPA) to its receptor (uPAR) in the local generation of plasmin by endothelium, we transfected spontaneously transformed immortalized human vascular endothelial cells that express high levels of uPA but low levels of uPAR with

Comparative evaluation of urokinase-type
✍ Alice Hemsen; Lutz Riethdorf; Nils BrΓΌnner; JΓΌrgen Berger; Sebastian Ebel; Chris πŸ“‚ Article πŸ“… 2003 πŸ› John Wiley and Sons 🌐 French βš– 233 KB πŸ‘ 1 views

## Abstract The urokinase‐type plasminogen activator receptor (uPAR, CD87) plays a central role in the plasminogen activation cascade, which participates in extracellular matrix degradation, cell migration and invasion. Here we performed a comprehensive immmunohistochemical evaluation of uPAR expre

Liver sinusoidal endothelial cells depen
✍ Kjetil Elvevold; Jaione Simon-Santamaria; Hege Hasvold; Peter McCourt; BΓ₯rd Smed πŸ“‚ Article πŸ“… 2008 πŸ› John Wiley and Sons 🌐 English βš– 528 KB

Liver sinusoidal endothelial cells (LSECs) are largely responsible for the removal of circulating lysosomal enzymes (LE) via mannose receptor (MR)-mediated endocytosis. We hypothesized that LSECs rely on this uptake to maintain their extraordinarily high degradation capacity for other endocytosed ma