𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Expression of PPARγ and Its Ligand-dependent Growth Inhibition in Human Brain Tumor Cell Lines

✍ Scribed by Kato, Mihoko ;Nagaya, Takashi ;Fujieda, Miyuki ;Saito, Kiyoshi ;Yoshida, Jun ;Seo, Hisao

Book ID
108582147
Publisher
Wiley (Blackwell Publishing)
Year
2002
Tongue
English
Weight
603 KB
Volume
93
Category
Article
ISSN
0910-5050

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

KLF4-dependent, PPARγ-induced expression
KLF4-dependent, PPARγ-induced expression of GPA33 in colon cancer cell lines
✍ Julie Rageul; Stéphanie Mottier; Anne Jarry; Yatrik Shah; Sandrine Théoleyre; Da 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 French ⚖ 272 KB

## Abstract The glycoprotein A33 (GPA33) is a colon cancer antigen. Phase I trials with ^131^I and ^125^I monoclonal antibody A33 in colon carcinoma patients showed excellent localization to colorectal cancer and some evidence of tumor response. Using DNA microarrays, we have identified the __GPA33

Identification of basic fibroblast growt
Identification of basic fibroblast growth factor sensitivity and receptor and ligand expression in human colon tumor cell lines
✍ Bonnie A. New; Lynn C. Yeoman 📂 Article 📅 1992 🏛 John Wiley and Sons 🌐 English ⚖ 868 KB

Basic fibroblast growth factor (bFGF) has been shown to be mitogenic to many different eukaryotic cell lines of mesodermal and neuroectoderrnal origin. Addition of exogenous bFCF to the chemically defined media of five characterized human colon tumor cell lines, cultured in the absence of epidermal

Compound 48, a novel dual PPAR α/γ ligan
Compound 48, a novel dual PPAR α/γ ligand, inhibits the growth of human CML cell lines and enhances the anticancer-effects of imatinib
✍ Janina Bertz; Chuanbing Zang; Hongyu Liu; Marlies Wächter; Kurt Possinger; H. Ph 📂 Article 📅 2009 🏛 Elsevier Science 🌐 English ⚖ 763 KB

Compound 48 (C48) is a novel dual ligand for peroxisome proliferator-activated receptor alpha and gamma (PPAR alpha/gamma). Culture of imatinib-sensitive and -resistant CML cell lines with C48 resulted in a strong growth inhibition which associated with G0/G1 cell cycle arrest. However, it showed no