𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Expression of HLA–B27 causes loss of migratory dendritic cells in a rat model of spondylarthritis

✍ Scribed by Lotta Utriainen; Dawn Firmin; Pamela Wright; Vuk Cerovic; Maxime Breban; Iain McInnes; Simon Milling


Book ID
115550501
Publisher
John Wiley and Sons
Year
2012
Tongue
English
Weight
581 KB
Volume
64
Category
Article
ISSN
0004-3591

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Defective costimulatory function is a st
✍ Cécile Hacquard-Bouder; Géraldine Falgarone; Antoine Bosquet; Faïza Smaoui; Domi 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 177 KB

## Abstract ## Objective A disease resembling the human spondylarthropathies develops in HLA–B27–transgenic rats. This disease in rats is mediated by CD4+ T cells, but antigen‐presenting cells (APCs) may also play a role. Dendritic cells (DCs) have been reported to be defective in allogeneic mixed

Switch of HLA-G alternative splicing in
✍ Nathalie Rouas-Freiss; Sylvie Bruel; Catherine Menier; Céline Marcou; Philippe M 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 French ⚖ 379 KB

Considerable information has been accumulated on HLA-G expression in tumor lesions in which HLA-G is viewed as a way to turn off anti-tumoral immunity. Nevertheless, there is little data concerning the mechanisms by which expression and function of HLA-G are regulated in malignant cells. Here, we ha