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Expression of Fas, Bcl-2 and p53 molecules in glomerulonephritis and their correlations with clinical and laboratory findings

✍ Scribed by AYSUN UĞUZ; GÜLFILIZ GÖNLÜŞEN; MELEK ERGIN; İLHAN TUNCER

Book ID
108958925
Publisher
John Wiley and Sons
Year
2005
Tongue
English
Weight
148 KB
Volume
10
Category
Article
ISSN
1320-5358

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## Abstract ## BACKGROUND. The p53 tumor suppressor gene product participated in G1 cell cycle arrest or cell death. Loss of function was associated with poor outcome in patients with breast carcinoma. __bcl__‐2 prevented apoptosis induced by c‐__myc__ or growth factor deprivation. High __bcl__‐2