The pathogenesis of pulmonary hypertension (PH) remains poorly understood. Vasoconstriction, although likely to be a major factor in the disease, varies between patients and studies of a variety of vasoactive substances have sometimes yielded conflicting results. Amongst these substances, alteration
Expression of endothelial nitric oxide synthase traffic inducer in the pladenta of pregnancy induced hypertension
β Scribed by Xiang Wenpei; Chen Hanping; Guo Yuzhen; Shen Hongling
- Publisher
- Springer-Verlag
- Year
- 2006
- Tongue
- Chinese
- Weight
- 267 KB
- Volume
- 26
- Category
- Article
- ISSN
- 0510-9752
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Nitric oxide (NOβ’), generated by nitric oxide synthase (NOS II) from immunostimulated cells during infection, plays an important role in host immune defense against microbial invasion. The impact of different rates of NOβ’ production on host cell function has not been defined. Herein, we describe the
Lipopolysaccharide (LPS) or a combination of interleukin (IL)-lp and interferon (1FN)-y cause transcriptional induction of a calcium-independent nitric oxide synthase (NOS) in astrocytes and C6 glioma cells. LPS induction of NOS in C6 cells was evidenced by a small amount of nitrite accumulation as