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Evidence against a role for endotoxin in the hepatic encephalopathy of rats with thioacetamide-induced fulminant hepatic failure

✍ Scribed by CHIEN-TING CHEN; CHI-JEN CHU; TEH-FANG WANG; REI-HWA LU; FA-YAUH LEE; FULL-YOUNG CHANG; HAN-CHIEH LIN; CHE-CHANG CHAN; SUN-SANG WANG; HUI-CHUN HUANG; SHOU-DONG LEE

Book ID
108950003
Publisher
John Wiley and Sons
Year
2005
Tongue
English
Weight
143 KB
Volume
20
Category
Article
ISSN
0815-9319

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## Abstract Increased ammonia is a major pathogenic factor in hepatic encephalopathy (HE), a neurologic syndrome associated with glutamatergic dysfunction. Previous studies have shown that in rat cerebral cortical slices or a glia‐derived cell line, acute treatment with ammonia in vitro and in vivo