𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Evaluation of recurring cytogenetic abnormalities in the treatment of myelodysplastic syndromes

✍ Scribed by Harold J. Olney; Michelle M. Le Beau


Publisher
Elsevier Science
Year
2007
Tongue
English
Weight
262 KB
Volume
31
Category
Article
ISSN
0145-2126

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


The treatment of the myelodysplastic syn
✍ Harvey D. Preisler πŸ“‚ Article πŸ“… 1999 πŸ› John Wiley and Sons 🌐 English βš– 67 KB πŸ‘ 2 views

T he article by Ferrara et al. 1 in this issue of Cancer describes the treatment of patients with poor prognosis myelodysplastic syndromes (MDS) using intensive remission induction chemotherapy followed by peripheral blood stem cell transplantation for patients age Ο½ 60 years. Thirty-two patients wi

Unique cytogenetic features of primary m
✍ Lin Li; Xu-Ping Liu; Ling Nie; Ming-Hua Yu; Yue Zhang; Tie-Jun Qin; Zhi-Jian Xia πŸ“‚ Article πŸ“… 2009 πŸ› Elsevier Science 🌐 English βš– 257 KB

Myelodysplastic syndromes (MDS) are a heterogeneous group of myeloid neoplasms. Chromosomal abnormalities have been detected in 40-70% patients with primary MDS and are heterogeneous among patients of different races and from different backgrounds. In the current study, 351 Chinese adult patients wi

The prognostic value of maturation-assoc
✍ I. Lorand-Metze; S.M.V. Califani; E. Ribeiro; C.S.P. Lima; K. Metze πŸ“‚ Article πŸ“… 2008 πŸ› Elsevier Science 🌐 English βš– 76 KB

Several phenotypic abnormalities of bone marrow (BM) hemopoietic precursors have been associated with disease progression in myelodysplastic syndromes (MDS). We analyzed the influence on overall survival of the expression of lineage and maturation-associated antigens of BM hemopoietic cells quantifi

Deletions of CDKN1B and ETV6 in acute my
✍ Patrik Andreasson; Bertil Johansson; Kristina Arheden; Rolf BillstrΓΆm; Felix Mit πŸ“‚ Article πŸ“… 1997 πŸ› John Wiley and Sons 🌐 English βš– 111 KB πŸ‘ 2 views

Seventy-nine acute myeloid leukemias (AML) and myelodysplastic syndromes without cytogenetic evidence of 12p aberrations were investigated by fluorescence in situ hybridization with probes for ETV6 and CDKN1B (previously called TEL and KIP1, respectively) to ascertain whether abnormalities of these