Epstein-barr virus subtype distribution in angioimmunoblastic lymphadenopathy
✍ Scribed by Bettina Borisch; Massimo Caioni; Nina Hurwitz; Corina Dommann-Scherrer; B. Odermatt; Ernst Waelti; R. Hubert Hubert; Rainer Kraft; Jean Laissue
- Publisher
- John Wiley and Sons
- Year
- 1993
- Tongue
- French
- Weight
- 679 KB
- Volume
- 55
- Category
- Article
- ISSN
- 0020-7136
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract Specific viral laboratory diagnosis of primary Epstein–Barr Virus (EBV) infection is usually based on antibody‐detection assays. During acute, lytic phase of infection, viral DNA can also be detected in serum. In the present study, the diagnostic utility of EBV DNA detection and quantit
Virus-like particles which seemed to be oncogenic have not been observed clinically in human neoplasms. However, we found some virus-like particles in the cytoplasm of a few proliferating cells in the lymph nodes of one patient with angioimmunoblastic lymphadenopathy. These were very similar to intr
## Abstract Expression of different panels of latent gene transcripts is controlled by usage of three distinct Epstein‐Barr virus (EBV) nuclear antigen (EBNA) promoters (Wp, Cp, and Qp). EBV‐associated hemophagocytic syndrome, which is often a fatal disease and generally occurs after primary EBV in
We examined Epstein-Barr virus (EBV)-specific antibodies in serum samples from 64 and 59 patients with EBV-positive and -negative gastric carcinomas, respectively, and 73 healthy controls using immunofluorescence assays. EBV capsid antigen (VCA) IgG and EBV-determined nuclear antigen (EBNA) IgG were