✦ LIBER ✦

(−)-Epigallocatechin gallate inhibits hepatitis C virus (HCV) viral protein NS5B

✍ Scribed by Changhyun Roh; Sung-Kee Jo

Book ID: 116905427
Publisher: Elsevier Science
Year: 2011
Tongue: English
Weight: 435 KB
Volume: 85
Category: Article
ISSN: 0039-9140
DOI: 10.1016/j.talanta.2011.08.035

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

(−)-Epigallocatechin-3-gallate inhibits

(−)-Epigallocatechin-3-gallate inhibits the replication cycle of hepatitis C virus

✍ Chao Chen, Hui Qiu, Jian Gong, Qing Liu, Han Xiao, Xin-Wen Chen, Bin-Lian Sun, R 📂 Article 📅 2012 🏛 Springer Vienna 🌐 English ⚖ 702 KB

Hepatitis C virus (HCV) NS2 protein up-r

Hepatitis C virus (HCV) NS2 protein up-regulates HCV IRES-dependent translation and down-regulates NS5B RdRp activity

✍ Yinglong She; Qingjiao Liao; Xulin Chen; Linbai Ye; Zhenghui Wu 📂 Article 📅 2008 🏛 Springer Vienna 🌐 English ⚖ 343 KB

The green tea polyphenol, epigallocatech

The green tea polyphenol, epigallocatechin-3-gallate, inhibits hepatitis C virus entry

✍ Sandra Ciesek; Thomas von Hahn; Che C. Colpitts; Luis M. Schang; Martina Friesla 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 708 KB

Effect of Hepatitis C Virus (HCV) NS5B-N

Effect of Hepatitis C Virus (HCV) NS5B-Nucleolin Interaction on HCV Replication with HCV Subgenomic Replicon

✍ Shimakami, T.; Honda, M.; Kusakawa, T.; Murata, T.; Shimotohno, K.; Kaneko, S.; 📂 Article 📅 2006 🏛 American Society for Microbiology 🌐 English ⚖ 287 KB

Hepatitis C virus NS2/3 protease regulat

Hepatitis C virus NS2/3 protease regulates HCV IRES-dependent translation and NS5B RdRp activity

✍ Yinglong She; Tao Han; Linbai Ye; Zhenghui Wu 📂 Article 📅 2009 🏛 Springer Vienna 🌐 English ⚖ 667 KB

Hepatitis C virus core, NS3, NS5A, NS5B

Hepatitis C virus core, NS3, NS5A, NS5B proteins induce apoptosis in mature dendritic cells

✍ Samila Siavoshian; Jean Daniel Abraham; Christine Thumann; Marie Paule Kieny; Ca 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 344 KB

Although reasons for hepatitis C virus (HCV) persistence are still unknown, specific cellular immune responses appear to influence the pathogenesis and outcome of the infection. Apoptosis of cells infected by viruses may appear suicidal to the viruses that induce programmed cell death of its host. H