Enhanced oxygen radical production in a transgenic mouse model of familial amyotrophic lateral sclerosis
β Scribed by Rugao Liu; John S. Althaus; Brenda R. Ellerbrock; David A. Becker; Dr. Mark E. Gurney
- Publisher
- John Wiley and Sons
- Year
- 1998
- Tongue
- English
- Weight
- 881 KB
- Volume
- 44
- Category
- Article
- ISSN
- 0364-5134
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Abstract ## Background and Objective Familial amyotrophic lateral sclerosis (FALS) is a neurodegenerative disease characterized by progressive loss of motor neurons and death. Mitochondrial dysfunction and oxidative stress play an important role in motor neuron loss in ALS. Light therapy (LT) h
## Abstract Amyotrophic lateral sclerosis (ALS) is a lateβonset progressive neurodegenerative disease affecting motor neurons. About 2% of patients with the disease are associated with mutations in the gene encoding Cu/Zn superoxide dismutase (SOD1). The purpose of this study is to assess the effec