๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Emerging genes associated with the progression of nonalcoholic fatty liver disease

โœ Scribed by Christina Koutsari; Konstantinos N. Lazaridis


Publisher
John Wiley and Sons
Year
2010
Tongue
English
Weight
138 KB
Volume
52
Category
Article
ISSN
0270-9139

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Progress in understanding the pathogenes
โœ Richard H. Moseley ๐Ÿ“‚ Article ๐Ÿ“… 2005 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 159 KB ๐Ÿ‘ 2 views

The pathogenesis of nonalcoholic steatohepatitis (NASH) is poorly defined. Feeding mice a diet deficient in methionine and choline (MCD diet) induces experimental NASH. Osteopontin (OPN) is a Th1 cytokine that plays an important role in several fibroinflammatory diseases. We examined the role of OPN

Bioinformatics as tool to identify gene/
โœ Anna Alisi; Matilde Marcellini; Valerio Nobili ๐Ÿ“‚ Article ๐Ÿ“… 2007 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 44 KB ๐Ÿ‘ 1 views

Non-alcoholic fatty liver disease (NAFLD) includes a wide spectrum of liver diseases ranging from simple hepatic steatosis to nonalcoholic steatohepatitis (NASH). 1,2 Because the prevalence of these diseases is increasing, determination of early diagnostic markers as well as development of a more ef

Nonalcoholic fatty liver disease: is it
โœ Dawn McDowell Torres; Stephen A. Harrison ๐Ÿ“‚ Article ๐Ÿ“… 2010 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 292 KB ๐Ÿ‘ 1 views

Background: Patients with cirrhosis in Child-Pugh class C or those in class B who have persistent bleeding at endoscopy are at high risk for treatment failure and a poor prognosis, even if they have undergone rescue treatment with a transjugular intrahepatic portosystemic shunt (TIPS). This study ev

Association of nonalcoholic fatty liver
โœ Menha Swellam; Nadia Hamdy ๐Ÿ“‚ Article ๐Ÿ“… 2012 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 239 KB

## Abstract Leptin (Lep), a 16โ€kDa polypeptide hormone, exerts its action through the leptin receptor (LepRb), a member of the class I cytokine receptor family. Both leptin and LepRb probably have been implicated in pathogenesis of nonalcoholic fatty liver disease (NAFLD). This study was designed t