𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Effects of testosterone, testosterone propionate, 17β-trenbolone and progesterone on cell transformation and mutagenesis in Syrian hamster embryo cells

✍ Scribed by Tsutsui, Takeki; Komine, Akiko; Huff, James; Barrett, J.Carl

Book ID
121695157
Publisher
Oxford University Press
Year
1995
Tongue
English
Weight
395 KB
Volume
16
Category
Article
ISSN
0143-3334

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Involvement of genotoxic effects in the
Involvement of genotoxic effects in the initiation of estrogen-induced cellular transformation: Studies using Syrian hamster embryo cells treated with 17β-estradiol and eight of its metabolites
✍ Takeki Tsutsui; Yukiko Tamura; Eiichi Yagi; J. Carl Barrett 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 French ⚖ 114 KB 👁 2 views

To examine a direct involvement of genotoxic effects of estrogens in the initiation of hormonal carcinogenesis, the abilities of 17beta-estradiol (E2) and 8 of its metabolites to induce cellular transformation and genetic effects were studied using the Syrian hamster embryo (SHE) cell model. Treatme

Effect of 17-β-estradiol and testosteron
Effect of 17-β-estradiol and testosterone on aryl hydrocarbon hydroxylase activity in mouse tissues in vivo and in cell culture
✍ D. W. Nebert; L. L. Bausserman; R. R. Bates 📂 Article 📅 1970 🏛 John Wiley and Sons 🌐 French ⚖ 895 KB

## Abstract Initiation of skin tumors by 7,12‐dimethylbenz[a]anthracene is more effective during diestrus than during estrus and thus may be inhibited by estrogens. We therefore studied the effect of sex hormones on aryl hydrocarbon hydroxylase, an enzyme system which may be a common pathway in met

Effects of a catechol-O-methyltransferas
Effects of a catechol-O-methyltransferase inhibitor on catechol estrogen-induced cellular transformation, chromosome aberrations and apoptosis in Syrian hamster embryo cells
✍ Yutaka Hirose; Takeo W. Tsutsui; Maki Ohno; J. Carl Barrett; Takeki Tsutsui 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 French ⚖ 260 KB

## Abstract To examine a possible mechanism of endogenous estrogen‐induced carcinogenesis, we studied the effect of the catechol‐__O__‐methyltransferase (COMT) inhibitor Ro 41‐0960 on cell transforming and clastogenic activities of 2 catechol estrogens 2‐ and 4‐hydroxyestrone (2‐ or 4‐OHE~1~) using