𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Effects of neurotoxic lesions in histaminergic neurons on brain tumor necrosis factor levels

✍ Scribed by X. A. Alvarez; A. Franco; L. Fernández-Novoa; R. Cacabelos


Publisher
SP Birkhäuser Verlag Basel
Year
1994
Tongue
English
Weight
236 KB
Volume
41
Category
Article
ISSN
1420-908X

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Neuronal death in cytokine-activated pri
✍ Martha Downen; Terry D. Amaral; Liwei L. Hua; Meng-Liang Zhao; Sunhee C. Lee 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 696 KB

We examined cytokine-mediated neuronal death in neuron-astrocyte cultures from second trimester human fetal cerebrum. In these cultures, high-output inducible nitric oxide synthase (NOS) and tumor necrosis factor-␣ (TNF␣) are expressed in astrocytes after exposure to IL-1␤/IFN␥. Neuronal cell death

Serum levels of soluble tumor-necrosis-f
✍ Magdalena Malejczyk; Jaroslaw Jóźwiak; Anna Osiecka; Piotr I. Roszkowski; Walent 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 French ⚖ 83 KB 👁 2 views

The levels of type-I and type-II soluble TNF-␣ receptors (sTNF-Rs) were evaluated in sera from patients with various human-papillomavirus-(HPV)-associated benign and malignant anogenital lesions using specific enzyme-linked immunobiological assays. In patients with benign HPV6/11-associated condylom

Neurotoxic effects of (±)fenfluramine an
✍ Una D. McCann; Jie Yuan; George A. Ricaurte 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 151 KB 👁 2 views

Until recently, (Ϯ)fenfluramine (FEN) was widely prescribed as an appetite suppressant. In animals, FEN is a potent and selective brain serotonin neurotoxin. The present studies assessed the effects of phentermine (PHEN), an appetite suppressant frequently used clinically in combination with FEN, on

Effect of recombinant human tumor necros
✍ E. Knippel; J. Rychly; H. A. Schulze; B. Ringel; A. Krygier-Stojalowska 📂 Article 📅 1988 🏛 John Wiley and Sons 🌐 French ⚖ 580 KB

The ability of recombinant human tumor necrosis factor (rH-TNF-alpha) to induce regression of sarcoma 180 in vivo was evaluated. The tumor was cured by TNF in the course of 4 weeks. TNF inhibited proliferation of sarcoma 180 cells in vitro, which suggests a direct effect of TNF on tumor cells in viv