Early life, the epigenome and human health

The early life environment and the epige

The early life environment and the epigenome

✍ Moshe Szyf 📂 Article 📅 2009 🏛 Elsevier Science 🌐 English ⚖ 354 KB

[Epigenetics and Human Health] Environme

[Epigenetics and Human Health] Environmental Epigenomics in Health and Disease || Developmental Epigenomics and Metabolic Disease

✍ Jirtle, Randy L; Tyson, Frederick L. 📂 Article 📅 2013 🏛 Springer Berlin Heidelberg 🌐 German ⚖ 148 KB

There are now compelling human epidemiological and animal experimental data that indicate the risk of developing adult-onset complex diseases and neurological disorders are influenced by persistent epigenetic adaptations in response to prenatal and early postnatal exposures to environmental factors.

Environmental epigenomics in human healt

Environmental epigenomics in human health and disease

✍ Dana C. Dolinoy; Randy L. Jirtle 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 149 KB

## Abstract The epigenome consists of the DNA methylation marks and histone modifications involved in controlling gene expression. It is accurately reproduced during mitosis and can be inherited transgenerationally. The innate plasticity of the epigenome also enables it to be reprogrammed by nutrit

Early Life Origins of Human Health and D

Early Life Origins of Human Health and Disease

✍ Rolf Zetterström 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 75 KB

Early Life Origins of Human Health and D

Early Life Origins of Human Health and Disease

✍ MIDORI AWAZU 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 91 KB

[Epigenetics and Human Health] Environme

[Epigenetics and Human Health] Environmental Epigenomics in Health and Disease || The Nature of Human Transgenerational Responses

✍ Jirtle, Randy L; Tyson, Frederick L. 📂 Article 📅 2013 🏛 Springer Berlin Heidelberg 🌐 German ⚖ 399 KB

There are now compelling human epidemiological and animal experimental data that indicate the risk of developing adult-onset complex diseases and neurological disorders are influenced by persistent epigenetic adaptations in response to prenatal and early postnatal exposures to environmental factors.