Dose increase augments response rate to interferon-α in chronic hepatitis C
✍ Scribed by Peter Ferenci; Rudolf Stauber; Albert Propst; Richard Fiedler; Christian Müller; Michael Gschwantler; Kurt Schütze; Christian Datz; Gerd Judmaier; Wolfgang Vogel; Günther J. Krejs; Alfred Gangl
- Book ID
- 105457780
- Publisher
- Springer US
- Year
- 1996
- Tongue
- English
- Weight
- 635 KB
- Volume
- 41
- Category
- Article
- ISSN
- 0163-2116
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Tumor necrosis factor a (TNF-a) is a cytokine with infectious agents and has been shown to exert direct pleiotropic properties that is induced in a variety of antiviral effects. [3][4][5] Although low levels of TNF-a can pathological situations including viral infections. In this contribute to cell
The likelihood of a sustained response to a course of interferon in patients with chronic hepatitis C correlates with several clinical and viral factors, including age, viral genotype and initial levels of hepatitis C virus (HCV) RNA in serum. The role of race and ethnicity has not been assessed. We