✦ LIBER ✦

DNA methylation at promoter CpG-rich sites regulates alkaline phosphatase expression

✍ Scribed by J. Delgado-Calle; J. Arozamena; C. Sañudo; V. Mijares; J.A. Riancho

Book ID: 116324329
Publisher: Elsevier Science
Year: 2011
Tongue: English
Weight: 63 KB
Volume: 48
Category: Article
ISSN: 8756-3282
DOI: 10.1016/j.bone.2011.03.194

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

DNA methylation at 5-HT1A receptor promo

DNA methylation at 5-HT1A receptor promoter C(− 1019)G polymorphism CpG sites in schizophrenia and depression

✍ M. Czesak; J. Lu; C.A. Stockmeier; M.C. Austin; H.Y. Meltzer; P.R. Albert⁎ 📂 Article 📅 2008 🏛 Elsevier Science 🌐 English ⚖ 58 KB

Epigenetic regulation of metallothionein

Epigenetic regulation of metallothionein-i gene expression: Differential regulation of methylated and unmethylated promoters by DNA methyltransferases and methyl CpG binding proteins

✍ Sarmila Majumder; Huban Kutay; Jharna Datta; Dennis Summers; Samson T. Jacob; Ka 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 623 KB

## Abstract Metallothioneins (MTs) are a group of cysteine‐rich stress response proteins that scavenge reactive oxygen species and heavy metals. Recently, we have shown that MT‐I promoter is methylated and suppressed in some solid and liquid tumors and can be robustly activated following treatment

Oxygen-regulated erythropoietin gene exp

Oxygen-regulated erythropoietin gene expression is dependent on a CpG methylation-free hypoxia-inducible factor-1 DNA-binding site

✍ Roland H. Wenger; Ivica Kvietikova; Andreas Rolfs; Gieri Camenisch; Max Gassmann 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 526 KB

Methylation of specific CpG sites in the

Methylation of specific CpG sites in the promoter region could significantly down-regulate p16INK4a expression in gastric adenocarcinoma

✍ Sang Hyun Song; Hyun-Soon Jong; Hyun Ho Choi; Shin Hyeok Kang; Min Hee Ryu; Noe 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 French ⚖ 228 KB 👁 1 views

## Silencing of p16 INK4a by methylation of the CpG islands in the promoter region has been found to be an alternative mechanism of inactivation in several tumors. However, in gastric carcinoma, the relationship between methylation status and the transcriptional silencing of the p16 gene remains t