Model for End-Stage Liver Disease (MELD) score-based allocation systems have been adopted by most countries in Europe and North America. Indeed, the MELD score is a robust marker of early mortality for patients with cirrhosis. Except for extreme values, high pretransplant MELD scores do not signific
Disease gravity and urgency of need as guidelines for liver allocation
β Scribed by Bijan Eghtesad; Oscar Bronsther; William Irish; Adrian Casavilla; Kareem Abu-Elmagd; David Van Thiel; Andreas Tzakis; John J. Fung; Thomas E. Starzl
- Publisher
- John Wiley and Sons
- Year
- 1994
- Tongue
- English
- Weight
- 746 KB
- Volume
- 20
- Category
- Article
- ISSN
- 0270-9139
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Determining need for liver transplantation (LT) can be effectively estimated when the actual liver disease is highly likely to cause death in the near future. However, for many conditions, the liver disease itself does not carry a high risk of short-term mortality, and other factors contribute to de
This guideline has been approved by the American Association for the Study of Liver Diseases (AASLD) and represents the position of the association. ## Preamble These recommendations provide a data-supported approach to establishing guidelines. They are based on the following: (1) a formal review
We examined the impact of the Model for End-Stage Liver Disease (MELD) organ allocation scheme on 44 patients with hepatocellular carcinoma (HCC) awaiting orthotopic liver transplantation (OLT) between February 2002 and January 2003, and compared the outcome with 58 patients listed in the 4 years be
The selection of living donor liver transplantation (LDLT) recipients in regions where deceased donor liver transplantation (DDLT) is rarely performed might be different from that in other centers at which LDLT is an alternative option to DDLT. Records of adult (age > or = 18 yr) patients referred t
The management of coagulopathy in patients with acute and chronic liver disease has undergone little change in many years despite advances in our understanding of the pathogenesis of this problem. In general, deficiency of clotting factors as a result of poor hepatic synthetic function accounts for