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Dihydromyricetin Ameliorates Behavioral Deficits and Reverses Neuropathology of Transgenic Mouse Models of Alzheimer’s Disease

✍ Scribed by Liang, Jing; Kerstin Lindemeyer, A.; Shen, Yi; López-Valdés, Héctor E.; Martínez-Coria, Hilda; Shao, Xuesi M.; Olsen, Richard W.

Book ID: 125366835
Publisher: Springer
Year: 2014
Tongue: English
Weight: 844 KB
Volume: 39
Category: Article
ISSN: 0364-3190
DOI: 10.1007/s11064-014-1304-4

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## Abstract The cerebral deposition of amyloid β‐peptide, a central event in Alzheimer's disease (AD) pathogenesis, begins several years before the onset of clinical symptoms. Noninvasive detection of AD pathology at this initial stage would facilitate intervention and enhance treatment success. In