Inhibition of the p38 pathway upregulate
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J. Perry Hall; Roger J. Davis
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Article
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2002
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John Wiley and Sons
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English
โ 456 KB
Mitogen-activated protein (MAP) kinases have been implicated as important mediators of the inflammatory response. Here we report that c-Jun NH(2)-terminal kinase (JNK), extracellular signal-regulated kinase (ERK), and p38 MAP kinase activities are reprogrammed during the IL-6 induced macrophage-like