𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Differential effects on nitric oxide synthase, heat shock proteins and glutathione in human endothelial cells exposed to heat stress and simulated diving

✍ Scribed by Fismen, Lise; Hjelde, Astrid; Svardal, Asbjørn M.; Djurhuus, Rune

Book ID
113041229
Publisher
Springer
Year
2011
Tongue
English
Weight
467 KB
Volume
112
Category
Article
ISSN
1439-6319

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Nitric oxide synthase induction in astro
Nitric oxide synthase induction in astroglial cell cultures: Effect on heat shock protein 70 synthesis and oxidant/antioxidant balance
✍ V. Calabrese; A. Copani; D. Testa; A. Ravagna; F. Spadaro; E. Tendi; V.G. Nicole 📂 Article 📅 2000 🏛 John Wiley and Sons 🌐 English ⚖ 208 KB 👁 2 views

Glial cells in the nervous system can produce nitric oxide in response to cytokines. This production is mediated by the inducible isoform of nitric oxide synthase. Radical oxygen species (ROS) and nitric oxide (NO) derivatives have been claimed to play a crucial role in many different processes, bot

Up-regulation of the association between
Up-regulation of the association between heat shock protein 90 and endothelial nitric oxide synthase prevents high glucose-induced apoptosis in human endothelial cells
✍ Lian-Yu Lin; Chien-Yu Lin; Fong-Ming Ho; Chiau S. Liau 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 216 KB 👁 1 views

## Abstract Hyperglycemia is the hallmark of diabetes mellitus. Poor glycemic control is correlated with increased cardiovascular morbidity and mortality. High glucose can trigger endothelial cell apoptosis by de‐activation of endothelial nitric oxide synthase (eNOS). eNOS was recently demonstrated