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Differential effects of ethanol on c-jun N-terminal kinase, 14-3-3 proteins, and Bax in postnatal day 4 and postnatal day 7 rat cerebellum

✍ Scribed by Marieta Barrow Heaton; Michael Paiva; Stacey Kubovic; Alexandra Kotler; Jonathan Rogozinski; Eric Swanson; Vladimir Madorsky; Michelle Posados

Book ID
113505917
Publisher
Elsevier Science
Year
2012
Tongue
English
Weight
980 KB
Volume
1432
Category
Article
ISSN
0006-8993

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## Abstract Exposure of the developing nervous system to ethanol (EtOH) produces neurological aberrations associated with fetal alcohol syndrome. During a well‐defined period, cerebellar neurons are highly susceptible to EtOH‐induced death, primarily through apoptosis. Neonatal rodent cerebellum is