𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Development of Novel C1 Domain Ligands of Protein Kinase C to Clarify the Precise Structure and Activation Mechanism

✍ Scribed by Go Hirai; Tadashi Shimizu; Mikiko Sodeoka


Publisher
John Wiley and Sons
Year
2006
Weight
8 KB
Volume
37
Category
Article
ISSN
0931-7597

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Challenges to the development of bryosta
✍ Kazuhiro Irie; Ryo C. Yanagita; Yu Nakagawa πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 314 KB

## Abstract Protein kinase C (PKC) isozymes are widely recognized as targets for anticancer therapy, and recent investigations demonstrated that PKC activators are potential therapeutic candidates for Alzheimer's disease and acquired immune deficiency syndrome. However, concerns exist about their t

A peptide fragment of ependymin neurotro
✍ David S. Adams; Brendan Hasson; Anne Boyer-Boiteau; Adam El-Khishin; Victor E. S πŸ“‚ Article πŸ“… 2003 πŸ› John Wiley and Sons 🌐 English βš– 376 KB πŸ‘ 1 views

## Abstract Ependymin (EPN) is a goldfish brain neurotrophic factor previously shown to function in a variety of cellular events related to long‐term memory formation and neuronal regeneration. CMX‐8933, an 8‐amino‐acid synthetic peptide fragment of EPN, was designed for aiding an investigation of

Diacylglycerol mediates the T-cell recep
✍ Rabindranath Chakrabarti; Sanjeev Kumar πŸ“‚ Article πŸ“… 2000 πŸ› John Wiley and Sons 🌐 English βš– 174 KB πŸ‘ 2 views

The mechanism of Ca 2Ο© influx in nonexcitable cells is not known yet. According to the capacitative hypothesis, Ca 2Ο© influx is triggered by IP 3 -mediated Ca 2Ο© release from the intracellular Ca 2Ο© stores. Conversely, many workers have reported a lack of association between release and influx. In t

Vitamin D receptor is not required for t
✍ Ramesh K. Wali; Juan Kong; Michael D. Sitrin; Marc Bissonnette; Yan Chun Li πŸ“‚ Article πŸ“… 2003 πŸ› John Wiley and Sons 🌐 English βš– 190 KB

## Abstract The rapid, non‐genomic actions of 1,25‐dihydroxyvitamin D~3~ [1,25(OH)~2~D~3~] have been well described, however, the role of the nuclear vitamin D receptor (VDR) in this pathway remains unclear. To address this question, we used VDR(+/+) and VDR(βˆ’/βˆ’) osteoblasts isolated from wild‐type