𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Development of hippocampal sclerosis after a complex febrile seizure

✍ Scribed by Andreas Merkenschlager; Horst Todt; Thomas Pfluger; Matthias K. Bernhard

Publisher
Springer
Year
2008
Tongue
English
Weight
184 KB
Volume
168
Category
Article
ISSN
0340-6997

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Relationship of hippocampal sclerosis to
Relationship of hippocampal sclerosis to duration and age of onset of epilepsy, and childhood febrile seizures in temporal lobectomy patients
✍ Keith G. Davies; Bruce P. Hermann; F.Curtis Dohan Jr.; Kevin T. Foley; Andrew J. πŸ“‚ Article πŸ“… 1996 πŸ› Elsevier Science 🌐 English βš– 581 KB

Controversy exists as to whether hippocampal sclerosis (HS) is a preexisting cause or a consequence of seizures. We investigated 122 consecutive patients who underwent anterior temporal lobectomy for intractable epilepsy between 1989 and 1992. MRI scans were normal apart from evidence of HS in 5 cas

Predictors of seizure freedom after surg
Predictors of seizure freedom after surgery for malformations of cortical development
✍ Edward F. Chang; Doris D. Wang; A. James Barkovich; Tarik Tihan; Kurtis I. Augus πŸ“‚ Article πŸ“… 2011 πŸ› John Wiley and Sons 🌐 English βš– 189 KB πŸ‘ 1 views

## Abstract ## Objective: Malformations of cortical development (MCDs) are a major cause of medically refractory epilepsy. Our aim was to examine a surgical series of patients with cortical malformations to determine the prognostic factors associated with long‐term seizure control. ## Methods: W

Development of a model of seizure-induce
Development of a model of seizure-induced hippocampal injury with features of programmed cell death in the BALB/c mouse
✍ Sachiko Shinoda; Tomohiro Araki; Jing-Quan Lan; Clara K. Schindler; Roger P. Sim πŸ“‚ Article πŸ“… 2004 πŸ› John Wiley and Sons 🌐 English βš– 645 KB

## Abstract Although mice are amenable to gene knockout, they have not been exploited in the setting of seizure‐induced neurodegeneration due to the resistance to injury of key mouse strains. We refined and developed models of seizure‐induced neuronal death in the C57BL/6 and BALB/c strains by foca