## Abstract Hepatitis B virus (HBV) DNA may persist in the liver in the absence of serum HBVβDNA after a selfβlimited acute hepatitis B. This may also occur in patients with chronic hepatitis C virus (HCV) infection but its prevalence and its impact on liver histology is unknown. HBVβDNA was tested
Detection of hepatitis B virus DNA levels in predicting the chronicity of acute viral hepatitis, type B
β Scribed by Shou-Dong Lee; Jaw-Ching Wu; Jiin-Yu Wang; Yang-Te Tsai; Kwang-Juei Lo; Benjamin N. Chiang
- Publisher
- John Wiley and Sons
- Year
- 1986
- Tongue
- English
- Weight
- 241 KB
- Volume
- 6
- Category
- Article
- ISSN
- 0270-9139
No coin nor oath required. For personal study only.
β¦ Synopsis
apologize for giving the wrong Rojkind reference. For historical interest, we used the 1976 (3) publication, but by error, referred to the 1979 one ( 1).
Dr. W. J. Lindblad makes an excellent point. The methods section was markedly abbreviated and the paper, as published, does give the misleading impression as though Type I collagen was reduced by P-mercaptoethanol. It just so happened that (J-mercaptoethanol was included in all the solutions, including the ones of Type I collagen which was reduced by heat. Figure 1 of our article (4) illustrates the migration of unreduced Type I nol. We are grateful to Dr. R. Timpl (Max Planck Institute in Munich, Federal Republic of Germany) for reviewing the electrophoretic patterns prior to publication and agreeing with the interpretation of the bands. REFERENCES 1 .
π SIMILAR VOLUMES
The aetiology of acute hepatitis in Taiwan: acute hepatitis superimposed on HBsAg carrier state as the main aetiology of acute hepatitis in areas with high HBsAg carrier rate. Infection 1988;
## Abstract To assess the pattern of development of serologic markers during acute hepatitis B, levels of HBsAg, HBeAg, and hepatitis B virus (HBV) DNA were assayed in stored serum samples obtained sequentially from 12 subjects infected with HBV during experimental studies conducted in the 1950s. S
Sera from 79 patients with acute self-limiting hepatitis, 17 patients with acute hepatitis B evolving into chronic HBsAg carriership, and 43 chronic HBsAg carriers without a history of acute hepatitis were analyzed for presence of hepatitis B virus (HBV)-DNA by a molecular hybridization technique. I
## DING-SHINN CHEN" During a follow-up period of 3.2 \* 1.6 (1 to 8.6) yr, 1,087 serum specimens from 230 HBsAg carrier children were tested for hepatitis B virus markers.