## Abstract In complex traits, multiple disease loci presumably interact to produce the disease. For this reason, even with high‐resolution single nucleotide polymorphism (SNP) marker maps, it has been difficult to map susceptibility loci by conventional locus‐by‐locus methods. Fine mapping strateg
✦ LIBER ✦
Detection of disease-associated deletions in case–control studies using SNP genotypes with application to rheumatoid arthritis
✍ Scribed by Chih-Chieh Wu; Sanjay Shete; Wei V. Chen; Bo Peng; Annette T. Lee; Jianzhong Ma; Peter K. Gregersen; Christopher I. Amos
- Publisher
- Springer
- Year
- 2009
- Tongue
- English
- Weight
- 619 KB
- Volume
- 126
- Category
- Article
- ISSN
- 0340-6717
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## Abstract ## Objective In Japanese individuals, the −169C/T single‐nucleotide polymorphism (SNP) in __FCRL3__ has been reported to be associated with rheumatoid arthritis (RA), systemic lupus erythematosus (SLE), and autoimmune thyroid diseases. The objective of this study was to test the associ
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