Depressed function of Kupffer cells in rats with CCl4-induced liver cirrhosis
β Scribed by S. Arii; K. Monden; S. Itai; T. Sasaoki; Y. Adachi; N. Funaki; H. Higashitsuji; T. Tobe
- Publisher
- Springer
- Year
- 1990
- Tongue
- English
- Weight
- 806 KB
- Volume
- 190
- Category
- Article
- ISSN
- 0372-8722
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
To investigate whether the impairment of mitochondrial function in cirrhosis is due to a reduction in liver cell mass or whether mitochondrial function is altered specifically, we analyzed mitochondrial volume and d a c e density of mitochondrial membranes in control and cirrhotic rate by stereologi
In cirrhosis, intrahepatic endothelial dysfunction is one of the mechanisms involved in the increased resistance to portal blood flow and therefore in the development of portal hypertension. Endothelial nitric oxide synthase (eNOS) uncoupling due to deficiency of tetrahydrobiopterin (BH4) results in