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Depletion of Arylhydrocarbon Receptor during Adipose Differentiation in 3T3-L1 Cells

โœ Scribed by Shigeki Shimba; Kohji Todoroki; Toshinori Aoyagi; Masakatsu Tezuka

Book ID
115582495
Publisher
Elsevier Science
Year
1998
Tongue
English
Weight
998 KB
Volume
249
Category
Article
ISSN
0006-291X

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## Abstract Tunicamycin, an antibiotic that inhibits protein glycosylation, elicited a rapid depletion of insulin binding activity at the surface of 3T3โ€L1 adipocytes. Disappearance of insulin receptors occurred more rapidly in the presence of tunicamycin than when protein synthesis was inhibited b