## Abstract Mice with deletions of nicotinic ACh receptor (nAChR) subunit genes are valuable models for studying nAChR functions. We could previously show in the mouse superior cervical ganglion (SCG) that the absence of distinct subunits affects the functional properties of receptors. Here, we hav
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Deletion of the beta 2 nicotinic acetylcholine receptor subunit alters development of tolerance to nicotine and eliminates receptor upregulation
โ Scribed by Sarah E. McCallum; Allan C. Collins; Richard Paylor; Michael J. Marks
- Publisher
- Springer
- Year
- 2005
- Tongue
- English
- Weight
- 307 KB
- Volume
- 184
- Category
- Article
- ISSN
- 0033-3158
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