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Deletion of TASK1 and TASK3 channels disrupts intrinsic excitability but does not abolish glucose or pH responses of orexin/hypocretin neurons

✍ Scribed by J. A. González; Lise T. Jensen; Susan E. Doyle; Manuel Miranda-Anaya; Michael Menaker; Lars Fugger; Douglas A. Bayliss; Denis Burdakov


Book ID
109027884
Publisher
John Wiley and Sons
Year
2009
Tongue
English
Weight
495 KB
Volume
30
Category
Article
ISSN
0953-816X

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