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Deficiency of inducible nitric oxide synthase exacerbates hepatic fibrosis in mice fed high-fat diet

✍ Scribed by Yi Chen; Shigenari Hozawa; Sadaaki Sawamura; Shinkichi Sato; Naoto Fukuyama; Chizuko Tsuji; Tetsuya Mine; Yasunori Okada; Ryuzaburo Tanino; Yoichi Ogushi; Hiroe Nakazawa

Book ID
116289536
Publisher
Elsevier Science
Year
2004
Tongue
English
Weight
865 KB
Volume
326
Category
Article
ISSN
0006-291X

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Lack of inducible nitric oxide synthase
Lack of inducible nitric oxide synthase leads to increased hepatic apoptosis and decreased fibrosis in mice after chronic carbon tetrachloride administration
✍ Ghazaleh Aram; James J. Potter; Xiaopu Liu; Michael S. Torbenson; Esteban Mezey πŸ“‚ Article πŸ“… 2008 πŸ› John Wiley and Sons 🌐 English βš– 696 KB

The role of nitric oxide (NO) in liver injury and fibrosis is unclear. The purpose of this study was to determine whether inducible NO synthase deficiency (iNOS(-/-)) affects liver injury and fibrosis produced in mice by chronic carbon tetrachloride (CCl(4)) administration. Wild-type (WT) or iNOS(-/