𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Defense of Living Body Against Oxidative Damage

✍ Scribed by Kiyomi Kikugawa

Publisher
John Wiley and Sons
Year
2005
Weight
8 KB
Volume
36
Category
Article
ISSN
0931-7597

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Studies in neuronal ceroid lipofuscinosi
Studies in neuronal ceroid lipofuscinosis: Enzymes of liver and brain tissues involved in the defense against oxidative damage
✍ Hari S. Garg; Yogesh C. Awasthi; William A. Neff; Naseem H. Ansari; Dr. Satish K πŸ“‚ Article πŸ“… 1982 πŸ› John Wiley and Sons 🌐 English βš– 411 KB

## Abstract The levels and the nature of peroxidase isozymes in the liver and brain tissues of a patient with Batten‐Spielmeyer‐Vogts (BSV) syndrome were found to be comparable to those of normal controls, indicating that peroxidase deficiency is probably not the basic genetic defect in Battens dis

Instant coffee with high chlorogenic aci
Instant coffee with high chlorogenic acid levels protects humans against oxidative damage of macromolecules
✍ Christine Hoelzl; Siegfried KnasmΓΌller; Karl-Heinz Wagner; Leonilla Elbling; Wol πŸ“‚ Article πŸ“… 2010 πŸ› John Wiley and Sons 🌐 English βš– 166 KB

Coffee is among the most frequently consumed beverages. Its consumption is inversely associated to the incidence of diseases related to reactive oxygen species; the phenomenon may be due to its antioxidant properties. Our primary objective was to investigate the impact of consumption of a coffee con

Cytoprotection against oxidative stress-
Cytoprotection against oxidative stress-induced damage of astrocytes by extracellular ATP via P2Y1 receptors
✍ Youichi Shinozaki; Schuichi Koizumi; Seiichi Ishida; Jun-Ichi Sawada; Yasuo Ohno πŸ“‚ Article πŸ“… 2004 πŸ› John Wiley and Sons 🌐 English βš– 543 KB

## Abstract Oxidative stress is the main cause of neuronal damage in traumatic brain injury, hypoxia/reperfusion injury, and neurodegenerative disorders. Although extracellular nucleosides, especially adenosine, are well known to protect against neuronal damage in such pathological conditions, the