𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Defects in signal transduction caused by a T cell receptor β chain substitution

✍ Scribed by Toufic A. Reno; Steven Ley; Eiji Sugiyama; Alain Cantagrel; Richard Blumberg; Joseph Bonventre; Cox Terhorst; Edward T. H. Yeh


Publisher
John Wiley and Sons
Year
1990
Tongue
English
Weight
850 KB
Volume
20
Category
Article
ISSN
0014-2980

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Activation signal transduction by β1 int
✍ Shingo Nakayamada; Kazuyoshi Saito; Kazuhisa Nakano; Yoshiya Tanaka 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 272 KB 👁 1 views

## Abstract ## Objective Beta 1 integrin is a representative adhesion molecule for cell–cell and cell–extracellular matrix interactions, and it provides costimulatory signals to T cells. However, the relevance of β1 integrin to T cell activation in systemic lupus erythematosus (SLE) remains unclea

Inhibition of abnormal T cell developmen
✍ Katsuyuki Yui; Avinash Bhandoola; Shinji Komori; Makoto Katsumata; Mark I. Green 📂 Article 📅 1992 🏛 John Wiley and Sons 🌐 English ⚖ 975 KB

## Inhibition of abnormal T cell development and autoimmunity in gld mice by transgenic T cell receptor p chain* Mice homozygous for the gld (generalized lymphoproliferative disease) mutation develop systemic autoimmune disease and severe lymphadenopathy due to an age-related accumulation in the p

Extracellular matrix induced by TGFβ imp
✍ Anne Marie Gagnon; Josée Chabot; Dheerja Pardasani; Alexander Sorisky 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 223 KB 👁 1 views

When 3T3-L1 preadipose cells are exposed to transforming growth factor b (TGFb), they synthesize more extracellular matrix (ECM) and resist differentiationinducing stimuli. The mechanism by which ECM suppresses adipose cell differentiation (adipogenesis) remains unknown. Since adipogenesis is an ins