## Abstract Loosening is recognized as one of the primary sources of total hip replacement (THR) failure. In this study the influence of the bone surface texture on loosening of the cement/bone interface was studied. Model cemented hip replacements were prepared and subjected to cyclic loads that i
Creep dominates tensile fatigue damage of the cement–bone interface
✍ Scribed by Do-Gyoon Kim; Mark A. Miller; Kenneth A. Mann
- Publisher
- Elsevier Science
- Year
- 2004
- Tongue
- English
- Weight
- 697 KB
- Volume
- 22
- Category
- Article
- ISSN
- 0736-0266
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✦ Synopsis
Abstract
Fatigue damage from activities of daily living has been considered to be a major cause of aseptic loosening in cemented total hip arthroplasty. The cement–bone interface is one region where loosening could occur, but to date the fatigue response of the interface has not been examined. Cement–bone specimens were prepared from fresh frozen human cadaver tissue using simulated in vivo conditions. Tensile fatigue tests to failure were performed in an environmental chamber. Loss of specimen stiffness (stiffness damage) and permanent displacement after unloading (creep damage) were found in all specimens. At failure, creep damage accounted for the majority (79.9 ± 10.6%) of the total strain damage accumulation at failure (apparent strain, ε = 0.0114 ± 0.00488). A power law relationship between strain‐damage rate and time‐to‐failure showed that the strain‐damage rate was an excellent predictor of the fatigue life of the cement–bone interface. The S–N response of the interface was obtained as a function of the applied stress ratio and the initial apparent strain. The total motion between cement and bone (72.2 ± 29.8 μm) prior to incipient failure due to both stiffness and creep fatigue damage may be sufficient to result in fibrous tissue formation and contribute to eventual clinical loosening. © 2004 Orthopaedic Research Society. Published by Elsevier Ltd. All rights reserved.
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## Abstract Loss of fixation at the cement–bone interface is known to contribute to aseptic loosening, but little is known about the mechanical damage response of this interface. An in vitro study using cement–bone specimens subjected to shear fatigue loading was performed, and the progression of s