Correction: Gap-junction-mediated propagation and amplification of cell injury
✍ Scribed by Lin, Jane H.-C. ;Weigel, Helga ;Cotrina, Maria Luisa ;Liu, Shujun ;Bueno, Earl ;Hansen, Anker J. ;Hansen, Thomas W. ;Goldman, Steven ;Nedergaard, Maiken
- Book ID
- 109791614
- Publisher
- Nature Publishing Group
- Year
- 1998
- Tongue
- English
- Weight
- 105 KB
- Volume
- 1
- Category
- Article
- ISSN
- 1097-6256
- DOI
- 10.1038/3745
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## Abstract Dendritic cells (DCs) in culture express at least connexin43, a protein subunit of gap junctions, and form gap junction channels, which could be important for T‐cells activation. Here, we evaluated whether DCs express connexins in vivo and also to identify components of their microenvir
## Background: Gap junction intercellular channels are required for metabolic cooperation between cells and regulate normal tissue homeostasis by means of the transfer of small molecules between contacting cells. not surprisingly, the gap junction phenotype is frequently lost during carcinogenesis