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Conventional protein kinase C (PKC)-α and novel PKCε, but not -δ, increase the secretion of an N-terminal fragment of Alzheimer's disease amyloid precursor protein from PKC cDNA transfected 3Y1 fibroblasts

✍ Scribed by Tadatoshi Kinouchi; Hiroyuki Sorimachi; Kei Maruyama; Keiko Mizuno; Shigeo Ohno; Shoichi Ishiura; Koichi Suzuki


Book ID
115930770
Publisher
Elsevier Science
Year
1995
Tongue
English
Weight
822 KB
Volume
364
Category
Article
ISSN
0014-5793

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