Control of in vitro growth of viviparous embryo mutants of maize by abscisic acid
β Scribed by Robichaud, C. S. ;Wong, J. ;Sussex, I. M.
- Publisher
- John Wiley and Sons
- Year
- 1979
- Tongue
- English
- Weight
- 348 KB
- Volume
- 1
- Category
- Article
- ISSN
- 0192-253X
No coin nor oath required. For personal study only.
β¦ Synopsis
The plant hormone abscisic acid (ABA) is believed t o play a role in the onset of developmental arrest in seeds. Embryos of the viviparous mutants of Zea mays d o not undergo arrest but germinate directly on the ear. This study investigates the possibility that the mutants v p l , vp5, vp7, vp8, and vp9 are defective in some aspect of ABA action. Mutant and wild type embryos were removed from developing seeds at 18, 21, and 24 days after pollination and cultured aseptically on media containing a range of ABA concentrations. Seedlings were harvested after seven days when lengths and fresh and dry weights were recorded. The results indicate that these five viviparous mutants differ in their response t o ABA. Two mutants, vp5 and vp8, exhibit the same sensitivity t o growth inhibition by ABA as wild type. The remaining three mutants, however, manifest a range of decreased sensitivities with vpl being the least sensitive, followed by vp7 and vp9.
π SIMILAR VOLUMES
## Abstract Transforming growth factorβbeta (TGFβΞ²) autoregulates its expression in several mammalian cell types. We now report that addition of TGFβΞ²s 1, 2, and 3 to primary chicken embryo cells differentially affects expression of the messenger RNAs for the different TGFβΞ² isoforms depending on t
## Abstract Methylseleninic acid (MSeA) is a synthetic organoselenium form known to be effective against mammary carcinogenesis __in vivo__. Using the synchronized mouse mammary epithelial tumor cell (TM6) model, we have previously shown that 5 ΞΌM MSeA significantly inhibits cell growth and induces
It was shown previously that mouse bone marrow cells transformed by simian virus 40 (SV40) show a reversible cell density-dependent phenotypic transition between the nonmacrophage (rapidly growing) and the macrophage (stationary) states; cells in low-density cultures are in the growing phase, expres
Cultures of mouse macrophage cell lines transformed by wild-type or the tsA640 mutant of simian virus 40 (SV40) show a reversible phenotypic transition between the nonmacrophage (proliferating phase) and the macrophage (stationary phase) states (Takayama, 1980; Tanigawa et al., 1983). Distribution o