Contribution of hyperammonemia and inflammatory factors to cognitive impairment in minimal hepatic encephalopathy
✍ Scribed by Vicente Felipo; Amparo Urios; Encarna Montesinos; Inmaculada Molina; Maria L. Garcia-Torres; Miguel Civera; Juan A. Del Olmo; Joaquin Ortega; Jose Martinez-Valls; Miguel A. Serra; Norberto Cassinello; Abdallah Wassel; Esperanza Jordá; Carmina Montoliu
- Publisher
- Springer
- Year
- 2011
- Tongue
- English
- Weight
- 761 KB
- Volume
- 27
- Category
- Article
- ISSN
- 0885-7490
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
Patients with minimal hepatic encephalopathy (MHE) have attention, response inhibition, and working memory difficulties that are associated with driving impairment and high motor vehicle accident risk. Navigation is a complex system needed for safe driving that requires functioning working memory an
Studies of the pathogenesis of hepatic encephalopathy are hampered by the lack of a satisfactory animal model. We examined the neurological features of rats after bile duct ligation fed a hyperammonemic diet (BDL+HD). Six groups were studied: sham, sham pair-fed, hyperammonemic, bile duct ligation (
Minimal hepatic encephalopathy (MHE) has a negative effect on patients' daily functioning. Thus far, no study has investigated the effect of treatment-related improvement in cognitive functions on health-related quality of life (HRQOL). We measured psychometric performance by number and figure conne
The production of several inflammatory cytokines, such as murine macrophage inflammatory protein-2 (MIP-2), tumor necrosis factor (TNF), and interleukin (IL)-1, was investigated in response to respiratory syncytial virus (RSV) infection in a murine macrophage cell line, RAW264.7, with special refere