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Conditional Deletion of TrkB but Not BDNF Prevents Epileptogenesis in the Kindling Model

โœ Scribed by Xiao-Ping He; Robert Kotloski; Serge Nef; Bryan W Luikart; Luis F Parada; James O McNamara

Book ID
116781981
Publisher
Elsevier Science
Year
2004
Tongue
English
Weight
515 KB
Volume
43
Category
Article
ISSN
0896-6273

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Reduction of TrkB expression de novo in
Reduction of TrkB expression de novo in the adult mouse impairs epileptogenesis in the kindling model
โœ Robert Kotloski; James O. McNamara ๐Ÿ“‚ Article ๐Ÿ“… 2009 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 898 KB

## Abstract Elucidating the mechanisms of epileptogenesis in molecular terms can identify targets for therapies aimed at preventing epileptogenesis or limiting its progression. Genetic perturbations have implicated signaling by the neurotrophin, BDNF, and its receptor, TrkB, in limbic epileptogenes

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Corticosterone regulates expression of BDNF and trkB but not NT-3 and trkC mRNA in the rat hippocampus
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Corticosterone has profound effects on growth, differentiation, and synaptic transmission of hippocampal neurons by activation of mineralocorticoid receptors (MRs) and glucocorticoid receptors (GRs). In the present study we tested if neurotrophins can be implicated in these effects. For this purpose