𝔖 Bobbio Scriptorium
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Complex I and Parkinson's Disease

✍ Scribed by J. Timothy Greenamyre; Todd B. Sherer; Ranjita Betarbet; Alexander V. Panov

Book ID
111741259
Publisher
John Wiley and Sons
Year
2001
Tongue
English
Weight
163 KB
Volume
52
Category
Article
ISSN
1521-6543

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Radical formation site of cerebral complex I and Parkinson's disease
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## Abstract Paraquat was reduced to the paraquat radical via complex I in bovine cerebral mitochondria and accelerated lipid peroxidation. Thirty‐kilodalton subunit of complex I was considered to be the radical formation site, because of its marked destruction by the paraquat radical. The lipid per

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There is evidence for a 37% deficiency of complex I activity in Parkinson's disease (PD), which appears to be specific for PD amongst parkinsonian syndromes and selective for the substantia nigra within the central nervous system. Rat studies have shown that, in the context of a normal nigrostriatal