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Cloning and expression of the two new variants of Nav1.5/SCN5A in rat brain

✍ Scribed by Cheng-Tao Ren; Dong-Mei Li; Shao-Wu Ou; Yun-Jie Wang; Yi Lin; Zhi-Hong Zong; Masaki Kameyama; Asako Kameyama

Book ID
113075211
Publisher
Springer
Year
2012
Tongue
English
Weight
975 KB
Volume
365
Category
Article
ISSN
0300-8177

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## Abstract Voltage‐gated sodium channels play critical roles in the excitability of the brain. A decreased level of Na~v~1.1 has been identified as the cause of severe myoclonic epilepsy in infancy. In the present study, we identified the transcription start site and three 5′‐untranslated exons of