𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Clear cells carcinoma of fallopian tubes associated with tubal endometriosis

✍ Scribed by F. J. de la Torre; F. Rojo; A. García

Publisher
Springer
Year
2002
Tongue
English
Weight
83 KB
Volume
266
Category
Article
ISSN
0003-9128

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Enhanced expression of Annexin A4 in cle
Enhanced expression of Annexin A4 in clear cell carcinoma of the ovary and its association with chemoresistance to carboplatin
✍ Ayako Kim; Takayuki Enomoto; Satoshi Serada; Yutaka Ueda; Tsuyoshi Takahashi; Ba 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 French ⚖ 247 KB 👁 1 views

## Abstract Clear cell carcinoma (CCC) of the ovary is known to be highly resistant to platinum‐based chemotherapy. The purpose of our study was to identify a candidate protein that is associated with chemoresistance of CCC and to investigate the specific mechanism of chemoresistance conferred by t

Low CAIX expression and absence of VHL g
Low CAIX expression and absence of VHL gene mutation are associated with tumor aggressiveness and poor survival of clear cell renal cell carcinoma
✍ Jean-Jacques Patard; Patricia Fergelot; Pierre I. Karakiewicz; Tobias Klatte; Qu 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 French ⚖ 150 KB 👁 1 views

## Abstract We attempted to describe, in a series of clear cell renal cell carcinoma (RCC), the relationship between CAIX expression, VHL gene mutations, tumor characteristics and outcome. Radical nephrectomy was performed in 100 patients. Genomic DNA was extracted from frozen tumor samples. Four a

Low frequency of HLA haplotype loss asso
Low frequency of HLA haplotype loss associated with loss of heterozygocity in chromosome region 6p21 in clear renal cell carcinomas
✍ Isabel Maleno; Miguel Angel Lopez Nevot; Barbara Seliger; Federico Garrido 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 French ⚖ 94 KB 👁 1 views

## Abstract HLA class I loss or downregulation is a widespread mechanism used by tumor cells to avoid tumor recognition by cytotoxic T lymphocytes favoring tumor immune escape. Multiple molecular mechanisms are responsible for these altered HLA class I tumor phenotypes. It has been described in dif