✦ LIBER ✦

Class I PI3K in oncogenic cellular transformation

✍ Scribed by Zhao, L; Vogt, P K

Book ID: 109970569
Publisher: Nature Publishing Group
Year: 2008
Tongue: English
Weight: 435 KB
Volume: 27
Category: Article
ISSN: 0950-9232
DOI: 10.1038/onc.2008.244

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Oncogenic signaling of class I PI3K isof

Oncogenic signaling of class I PI3K isoforms

✍ Denley, A; Kang, S; Karst, U; Vogt, P K 📂 Article 📅 2007 🏛 Nature Publishing Group 🌐 English ⚖ 678 KB

An essential role for protein synthesis

An essential role for protein synthesis in oncogenic cellular transformation

✍ Bader, Andreas G; Vogt, Peter K 📂 Article 📅 2004 🏛 Nature Publishing Group 🌐 English ⚖ 110 KB

IL-10 inhibits the starvation induced au

IL-10 inhibits the starvation induced autophagy in macrophages via class I phosphatidylinositol 3-kinase (PI3K) pathway

✍ Hun-Jung Park; Suk Jun Lee; Sang-Hoon Kim; Jihye Han; Joonbeom Bae; Sang Joon Ki 📂 Article 📅 2011 🏛 Elsevier Science 🌐 English ⚖ 922 KB

Metformin rescues cell surface major his

Metformin rescues cell surface major histocompatibility complex class I (MHC-I) deficiency caused by oncogenic transformation

✍ Oliveras-Ferraros, Cristina; Cufí, Sílvia; Vazquez-Martin, Alejandro; Menendez, 📂 Article 📅 2012 🏛 Landes Bioscience 🌐 English ⚖ 650 KB

A class IA PI3K controls inflammatory cy

A class IA PI3K controls inflammatory cytokine production in human neutrophils

✍ Carl F. Fortin; Alexandre Cloutier; Thornin Ear; Stéphanie Sylvain-Prévost; Thom 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 372 KB

Down-regulation of the MHC class I antig

Down-regulation of the MHC class I antigen-processing machinery after oncogenic transformation of murine fibroblasts

✍ Barbara Seliger; Christina Harders; Sabine Lohmann; Frank Momburg; Stefanie Urli 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 255 KB

Malignant transformation is often associated with genetic alterations providing tumor cells with mechanisms for escape from immune surveillance. Human and murine tumors of various origin as well as in vitro models of viral and oncogenic transformation express reduced levels of major histocompatibili