Some liver allograft recipients with hepatitis C virus (HCV) infection develop hyperbilirubinemia, which might be the result of a cholestatic variant of hepatitis C. We evaluated all liver biopsy samples from 6 liver transplant recipients who had polymerase chain reaction-positive HCV infection and
Chronic hepatitis in liver allografts
β Scribed by Stefan G. Hubscher; Mrcpath; Raouf E. Nakhleh; Dale C. Snover
- Publisher
- John Wiley and Sons
- Year
- 1990
- Tongue
- English
- Weight
- 176 KB
- Volume
- 12
- Category
- Article
- ISSN
- 0270-9139
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π SIMILAR VOLUMES
1. Hepatitis C virus (HCV) RNA+ liver allograft recipients invariably reinfect the liver allograft within hours after transplantation, and the majority (>70%) develop chronic hepatitis. The rate at which these patients experience progression to cirrhosis and the overall percentage are significantly
micrometers thick) were cut, fixed, and stored at 070ΠC.
In this issue, Fiel et al. 1 use the phrase "plasma cell hepatitis (de novo autoimmune hepatitis)" to describe what they eventually conclude to be a variant of acute rejection. This study was likely prompted by curiosity about the significance of plasma cell-rich infiltrates in liver allograft biops
The CD40-CD40L (CD154) interaction plays a pivotal role in the effector mechanisms of allograft rejection. Blockade of the CD40/CD40L costimulatory pathway prevents the development of chronic allograft rejection in several animal transplant models. The relevance of in situ CD40 and CD40L expression