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Choline exposure reduces potentiation of N-methyl-d-aspartate toxicity by corticosterone in the developing hippocampus

✍ Scribed by Patrick J. Mulholland; Rachel L. Self; Barton R. Harris; John M. Littleton; Mark A. Prendergast


Book ID
113567090
Publisher
Elsevier Science
Year
2004
Tongue
English
Weight
314 KB
Volume
153
Category
Article
ISSN
0165-3806

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## Abstract Zinc is an endogenous N‐methyl‐D‐aspartate (NMDA) receptor blocker. It is possible that zinc‐mediated modification of hippocampal CA1 long‐term potentiation (LTP) is linked to the expression of NMDA receptor subunits, which varies with postnatal development. In the present study, the ef