𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Characterization of a mutation that causes overproduction of inositol in Neurospora crassa

✍ Scribed by Schablik, Marcella ;Kiss, Akos ;Zsindley, Attila ;Szabó, Gábor


Publisher
Springer
Year
1988
Tongue
English
Weight
456 KB
Volume
213
Category
Article
ISSN
0026-8925

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


KMeyeDB: a graphical database of mutatio
✍ Takashi Kawamura; Masafumi Ohtsubo; Susumu Mitsuyama; Saho Ohno-Nakamura; Nobuyo 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 429 KB

KMeyeDB (http://mutview.dmb.med.keio.ac.jp/) is a database of human gene mutations that cause eye diseases. We have substantially enriched the amount of data in the database, which now contains information about the mutations of 167 human genes causing eyerelated diseases including retinitis pigment

Genetic characterization of a mutation t
✍ L. G. Hickok; O. J. Schwarz 📂 Article 📅 1989 🏛 Springer 🌐 English ⚖ 401 KB

Three nuclear mutations that affect tolerance to the herbicide paraquat have been selected in the fern Ceratopteris richardii. Two of the mutations, pq2 and pq45 are allelic and confer low and moderate tolerance, respectively. A third mutation, pqa6, is not linked to the other two and significantly

A screen for mutations that prevent leth
✍ Maixner, Andrew ;Hecker, Timothy P. ;Phan, Quang N. ;Wassarman, David A. 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 305 KB

Ras1 plays a critical role in receptor tyrosine kinase (RTK) signal transduction pathways that function during Drosophila development. We demonstrate that mis-expression of constitutively active forms of Ras1 (Ras1V12) and the Sevenless (Sev) RTK (SevS11) during embryogenesis causes lethality due to

Identification and characterization of S
✍ Laura Rodríguez-Pascau; Laura Gort; Edward H. Schuchman; Lluïsa Vilageliu; Danie 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 195 KB

Niemann-Pick disease (NPD) types A/B are both caused by a deficiency of lysosomal acid sphingomyelinase and display autosomal recessive inheritance. These two types of the disease were described according to the presence (type A) or absence (type B) of neurological symptoms. We present a molecular a