𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Cell type-specific regulation of CCN2 protein expression by PI3K–AKT–FoxO signaling

✍ Scribed by Jana Samarin; Iwona Cicha; Margarete Goppelt-Struebe


Book ID
107594196
Publisher
Springer Netherlands
Year
2009
Tongue
English
Weight
180 KB
Volume
3
Category
Article
ISSN
1873-9601

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Down-regulation of MEK/ERK signaling by
✍ Patrick Laprise; Marie-Josée Langlois; Marie-Josée Boucher; Christian Jobin; Nat 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 261 KB 👁 2 views

In vitro experiments have shown that the establishment of cell-cell contacts in intestinal epithelial cell cultures is a critical step in initiating ERK inhibition, cell cycle arrest, and induction of the differentiation process. Herein, we determined the mechanisms through which E-cadherin-mediated

PI3K/AKT regulates aggrecan gene express
✍ Chin-Chang Cheng; Yoshiyasu Uchiyama; Akihiko Hiyama; Sachin Gajghate; Irving M. 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 414 KB

## Abstract The goal of the investigation was to test the hypothesis that the phosphoinositide‐3 kinase (PI3K)/AKT signaling pathway regulates the expression of the major extracellular matrix component of the intervertebral disc, aggrecan, in nucleus pulposus cells. Primary rat nucleus pulposus cel

PI3K signaling maintains c-myc expressio
✍ Carolin Schild; Matthias Wirth; Maximilian Reichert; Roland M. Schmid; Dieter Sa 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 339 KB

## Abstract Phosphatidylinositol 3‐kinase (PI3K) signaling controls survival and proliferation of cancer cells and is activated in around 60% of pancreatic ductal adenocarcinomas (PDACs). Although not entirely clarified, PI3K signaling is linked to cell cycle progression of PDAC cells. In this stud