𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Cartilage oligomeric matrix protein expression in hepatocellular carcinoma and the cirrhotic liver

✍ Scribed by YI XIAO; JÖRG KLEEFF; JUNCHAO GUO; AMIQ GAZDHAR; QUAN LIAO; PAUL E DI CESARE; Markus W Büchler; HELMUT FRIESS

Book ID
108949732
Publisher
John Wiley and Sons
Year
2004
Tongue
English
Weight
381 KB
Volume
19
Category
Article
ISSN
0815-9319

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Galectin-3 expression is induced in cirr
Galectin-3 expression is induced in cirrhotic liver and hepatocellular carcinoma
✍ Daniel K. Hsu; Christopher A. Dowling; K.-C. George Jeng; Jung-Ta Chen; Ri-Yao Y 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 French ⚖ 262 KB 👁 2 views

Galectins are a family of ␤-galactoside-binding animal lectins. In particular, a widely studied member galectin-3, previously designated as ⑀BP, CBP35, Mac-2, L-29 and L-34, has been associated with assorted processes such as cell growth, tumor transformation and metastasis. Galectin-3 is expressed

A silencer element in the cartilage olig
A silencer element in the cartilage oligomeric matrix protein gene regulates chondrocyte-specific expression
✍ Paul S Issack; Chuan-ju Liu; Lisa Prazak; Paul E Di Cesare 📂 Article 📅 2004 🏛 Elsevier Science 🌐 English ⚖ 774 KB

## Abstract The molecular mechanisms by which mesenchymal cells differentiate into chondrocytes are poorly understood. The cartilage oligomeric matrix protein gene (COMP) encodes a noncollagenous extracellular matrix protein whose expression pattern correlates with chondrocyte differentiation and a

Expression of mutant cartilage oligomeri
Expression of mutant cartilage oligomeric matrix protein in human chondrocytes induces the pseudoachondroplasia phenotype
✍ Thomas M. Merritt; Joseph L. Alcorn; Richard Haynes; Jacqueline T. Hecht 📂 Article 📅 2006 🏛 Elsevier Science 🌐 English ⚖ 404 KB

Over 70 mutations in the cartilage oligomeric matrix protein (COMP), a large extracellular pentameric glycoprotein synthesized by chondrocytes, have been identified as causing two skeletal dysplasias: multiple epiphyseal dysplasia (MED/EDM1), and a dwarfing condition, pseudoachondroplasia (PSACH). T