✦ LIBER ✦

Callosal agenesis and left ventricular hypertrabeculation/noncompaction

✍ Scribed by Josef Finsterer

Publisher: John Wiley and Sons
Year: 2010
Tongue: English
Weight: 46 KB
Volume: 152A
Category: Article
ISSN: 1552-4825
DOI: 10.1002/ajmg.a.33115

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Reply to Dr. Finsterer “Callosal Agenesi

Reply to Dr. Finsterer “Callosal Agenesis and Left Ventricular Hypertrabeculation/Noncompaction”

✍ Antonio Richieri-Costa; Dagma V.M. Abramides; Luciana P. Maximino; Celia M. Giac 📂 Article 📅 2010 🏛 John Wiley and Sons 🌐 English ⚖ 45 KB

Heterogenous myopathic background of lef

Heterogenous myopathic background of left ventricular hypertrabeculation/noncompaction

✍ Josef Finsterer; Claudia Stöllberger 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 33 KB

Left ventricular noncompaction in Sotos

Left ventricular noncompaction in Sotos syndrome

✍ Hugo R. Martinez; John W. Belmont; William J. Craigen; Michael D. Taylor; John L 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 116 KB 👁 1 views

Response to correspondence by Dr. Finste

Response to correspondence by Dr. Finsterer and Dr. Stöllberger: Heterogenous myopathic background of left ventricular hypertrabeculation/noncompaction

✍ Ludwig Thierfelder; Sabine Sasse-Klaassen; Brenda Gerull; Rolf Jenni; Erwin Oech 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 English ⚖ 34 KB

Possible new syndrome: Left ventricular

Possible new syndrome: Left ventricular noncompaction, partial agenesis of the corpus callosum, and developmental delay in a Brazilian child

✍ Dionisia A.C. Lamonica; Dagma V.M. Abramides; Luciana P. Maximino; Mariana G. Ge 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 145 KB 👁 2 views

Genetic heterogeneity of isolated noncom

Genetic heterogeneity of isolated noncompaction of the left ventricular myocardium

✍ Digilio, Maria Cristina; Marino, Bruno; Bevilacqua, Maurizio; Musolino, Anna Mar 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 4 KB 👁 2 views

Fig. 1. Pedigrees of two families of patients with INVM, showing recurrence of INVM in brother and sister in one family (A), and parental consanguinity in the other (B).