## Abstract Nitric oxide (NO) can regulate osteoblast activity. In this study, we evaluated the effects of pretreatment with a low concentration of NO on osteoblast injuries induced by a high level of NO and its possible molecular mechanisms. Exposure of osteoblasts to 0.3 mM sodium nitroprusside (
c-Jun N-terminal kinase and nuclear factor κB mediate nitric oxide-induced expression of matrix metalloproteinase-13
✍ Scribed by Lin Yang; Ai Guo; Jun-Chao Gu
- Publisher
- Springer
- Year
- 2010
- Tongue
- English
- Weight
- 385 KB
- Volume
- 35
- Category
- Article
- ISSN
- 0341-2695
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract The roles of the mitogen‐activated kinase protein (MAPK) pathway, nuclear factor‐kappa B (NF‐κB), and activator protein‐1 (AP‐1) in cellular responses to growth factors and mitogen are well established. However, the manner by which these proliferative pathways are affected by the tumor
Matrix metalloproteinase (MMP) plays an important role in the invasion and metastasis of cancer cells. The inhibitory effects of bergamottin, a cytochrome P450 inhibitor from Citrus paradis (grapefruit), on tumor invasion and migration and the possible mechanisms involved in this inhibition were inv
In response to the bacterial endotoxin, LPS, Kupffer cells are induced to express NO and TNF-␣. These compounds are involved in hepatic inflammation/injury, especially that associated with endotoxic shock. In this study, we demonstrate that ebselen (2-phenyl-1,2-benzisoselenazol-3[2H]one), a selenoo
## Abstract The original article to which this Erratum refers was published in __Molecular Carcinogenesis__ 2005;45:26–37.